Difference between revisions of "Balaban 2005 Cell"
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{{Publication | {{Publication | ||
|title=Balaban RS, Nemoto S, Finkel T (2005) Mitochondria, oxidants, and aging. Cell 120 | |title=Balaban RS, Nemoto S, Finkel T (2005) Mitochondria, oxidants, and aging. Cell 120:483-95. | ||
|info=[https://pubmed.ncbi.nlm.nih.gov/15734681/ PMID:15734681 Open Access] | |info=[https://pubmed.ncbi.nlm.nih.gov/15734681/ PMID:15734681 Open Access] | ||
|authors=Balaban RS, Nemoto S, Finkel T | |authors=Balaban RS, Nemoto S, Finkel T | ||
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|editor=Komlodi T | |editor=Komlodi T | ||
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Revision as of 22:09, 16 February 2021
| Balaban RS, Nemoto S, Finkel T (2005) Mitochondria, oxidants, and aging. Cell 120:483-95. |
Balaban RS, Nemoto S, Finkel T (2005) Cell
Abstract: The free radical theory of aging postulates that the production of intracellular reactive oxygen species is the major determinant of life span. Numerous cell culture, invertebrate, and mammalian models exist that lend support to this half-century-old hypothesis. Here we review the evidence that both supports and conflicts with the free radical theory and examine the growing link between mitochondrial metabolism, oxidant formation, and the biology of aging.
• Bioblast editor: Komlodi T
Cited by
- Komlódi T, Cardoso LHD, Doerrier C, Moore AL, Rich PR, Gnaiger E (2021) Coupling and pathway control of coenzyme Q redox state and respiration in isolated mitochondria. Bioenerg Commun 2021.3. https://doi.org/10.26124/bec:2021-0003
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